Author

Wei‐Yun Lai

University of GothenburgORCID 0000-0001-8515-1358Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:drug response / drug discovery· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Organism:human· 1 Research Focus:protein structure prediction· 1

Frequent Co-Authors

Bengt HallbergiD· 2 shared Jimmy Van den EyndeniD· 2 shared Marcus BorenäsiD· 2 shared Ruth Helen PalmeriD· 2 shared Agata AniszewskaiD· 1 shared Alexander SchmidtiD· 1 shared Anneli StrömbergiD· 1 shared Arne ClaeysiD· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersCancer Biology

Bengt HallbergORCID

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2 papers

Recent·Most cited

Cancer BiologyiSciencePeer-reviewedMar 5, 2026

RUVBL1 and RUVBL2 are druggable MYCN regulators in neuroblastoma.

Joachim Tetteh Siaw, Arne Claeys, Wei‐Yun Lai…·Jimmy Van den Eynden

This study identifies RUVBL1 and RUVBL2 as druggable regulators of MYCN in neuroblastoma, showing their inhibition reduces MYC signaling and induces cell cycle arrest and apoptosis.

DOI 10.1016/j.isci.2026.115236

mousehuman

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Cancer BiologyProceedings of the National Academy of SciencesPeer-reviewedDec 28, 2023

ALK signalling primes the DNA damage response sensitizing ALK-driven neuroblastoma to ATR inhibition

Marcus Borenäs, Ganesh Umapathy, Dan Emil Lind…·Ruth Helen Palmer

This study identifies ALK signaling's role in enhancing DNA damage response in neuroblastoma, showing that combined ATR/ALK inhibition leads to sustained tumor response and differentiation.

27 citations

mousebulk RNA-seq

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