Archita Das

This study identifies DRP1 sulfenylation as a redox-dependent mechanism driving reparative macrophage polarization and neovascularization in ischemic muscle.

This study shows that endothelial CTR1 senses disturbed flow by increasing mitochondrial copper, leading to cuproptosis and atherosclerosis, and identifies a therapeutic target.

This study investigates the role of copper transport proteins in Alzheimer's disease by examining their expression and function in AD mouse models and human brain endothelial cells.

This study shows that myeloid Drp1 deficiency impairs revascularization in ischemic muscle by promoting pro-inflammatory macrophage polarization and metabolic reprogramming.