Dipankar Ash

This study shows that CTR1 contributes to retinal ischemia-reperfusion injury by promoting oxidative stress and inflammation, and that copper chelators like TTM can mitigate these effects.

This study shows that endothelial CTR1 senses disturbed flow by increasing mitochondrial copper, leading to cuproptosis and atherosclerosis, and identifies a therapeutic target.

This study shows that myeloid Drp1 deficiency impairs revascularization in ischemic muscle by promoting pro-inflammatory macrophage polarization and metabolic reprogramming.