Author

Jin‐Sun Kim

Vanderbilt UniversityORCID 0000-0001-9382-7442Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 1 Assay:electrophysiology· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Research Focus:metabolism & bioenergetics· 1 Organism:mouse· 1

Frequent Co-Authors

Arya Y. NakheiD· 1 shared David Aaron JacobsoniD· 1 shared Jean‐Philippe CartailleriD· 1 shared Jordyn R. DobsoniD· 1 shared Leesa L. SampsoniD· 1 shared Mark A. MagnusoniD· 1 shared Matthew T. DickersoniD· 1 shared Prasanna K. DadiiD· 1 shared

Activity by Year

2023-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperPhysiology

Arya Y. NakheORCID

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1 paper

Recent·Most cited

PhysiologyeLifePeer-reviewedSep 20, 2023

The MODY-associated KCNK16 L114P mutation increases islet glucagon secretion and limits insulin secretion resulting in transient neonatal diabetes and glucose dyshomeostasis in adults

Arya Y. Nakhe, Prasanna K. Dadi, Jin‐Sun Kim…·David Aaron Jacobson

This study shows that the KCNK16 L114P mutation causes neonatal diabetes and glucose dyshomeostasis in adult mice by disrupting islet insulin secretion and increasing glucagon secretion.

9 citations

mouseelectrophysiology

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