Author

J. K. H. Tay

University of UtahORCID 0000-0002-0851-0537Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:genetic perturbation / CRISPR· 2 Assay:proteomics· 2 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Allie H. GrossmanniD· 2 shared Aaron Rogers· 1 shared Alex J. BottiD· 1 shared Alexis Bowles· 1 shared Austin Bender· 1 shared Chris StubbeniD· 1 shared Coulson P. Rich· 1 shared Emily C. Wilson· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersBiochemistry

Allie H. GrossmannORCID

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2 papers

Recent·Most cited

BiochemistryCell ReportsPeer-reviewedJan 1, 2025

SLC7A5 is required for citrulline-dependent growth in arginine limited conditions

K. N. Dunlap, Austin Bender, Alexis Bowles…·Gregory S. Ducker

This study identifies SLC7A5 as essential for citrulline-dependent growth in arginine-limited conditions and shows its role in citrulline uptake and tumor growth.

16 citationstop 3%

humanproteomics

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Cancer BiologyNature CommunicationsPeer-reviewedAug 4, 2024

ARF6-dependent endocytic trafficking of the Interferon-γ receptor drives adaptive immune resistance in cancer.

Yinshen Wee, Junhua Wang, Emily C. Wilson…·Allie H. Grossmann

This study identifies ARF6-dependent endocytic trafficking of the IFN-gamma receptor as a mechanism driving adaptive immune resistance in cancer, linking it to immune checkpoint blockade efficacy.

12 citations

mouseproteomics

𝕏 PostBluesky