Neil R. W. Martin

The paper shows human senescent myoblasts are more susceptible to reductive stress-induced cell death than proliferating cells, suggesting senolytic compounds may work via reductive stress.

This study identifies ROS-dependent Akt-mTORC1 dysregulation in senescent myoblasts and shows antioxidants can suppress inflammation and promote differentiation but also induce cell death.

This study examines the effects of monosodium iodoacetate (MIA) on skeletal muscle cell regeneration and hypertrophy in vitro, finding it delays regeneration and inhibits hypertrophy.