Daniela Strenkert

The paper compares transcriptomic responses to hyperoxia in two Chlamydomonas reinhardtii ecotypes, revealing distinct molecular adaptations.

This study shows that copper deficiency in Chlamydomonas reinhardtii increases SAD expression, leading to changes in fatty acid composition and growth, with FDX5 playing a role in electron transfer for lipid desaturation.

Depletion of chloroplast HSP70B causes cell division arrest, proteostasis collapse, impaired VIPP1 oligomer dynamics with aberrant thylakoid structures, and increased light sensitivity.

The paper quantifies protein abundances and stoichiometries in Chlamydomonas reinhardtii during the cell cycle using the total protein approach.

The paper investigates the functional distribution of FDX1 and FDX2 in Chlamydomonas, showing their distinct roles in carbon, nitrogen, and sulfur assimilation through mutant analysis.

The paper identifies ZNG3 as a Zn chaperone interacting with Cia5 to regulate Zn and CO2 metabolism in Chlamydomonas, with distinct phenotypes in ZNG3 and Cia5 mutants.

The paper identifies a mutation in CEHC1, which encodes FBXO3, leading to normoxic expression of HYDA genes in Chlamydomonas reinhardtii by stabilizing CRR1.

This study identifies distinct roles of CTR1 and CTR2 in Cu import and mobilization in Chlamydomonas, with CTR1 mediating high-affinity uptake and CTR2 facilitating Cu remobilization under Zn deficiency.

The paper presents version 6 of the Chlamydomonas reinhardtii reference genome, improving assembly quality and revealing structural mutations in laboratory strains.

The paper optimizes a ChIP-Seq framework for histone modification profiling in Chromochloris zofingiensis and identifies H3K4me3 enrichment at transcription start sites.

This study uses ChIP-Seq to profile H3K4 methylation across the Chlamydomonas cell cycle, revealing distinct patterns of mono- and trimethylation at gene promoters and their relationship to gene expression.