Stefan Schmollinger

This study shows that copper deficiency in Chlamydomonas reinhardtii increases SAD expression, leading to changes in fatty acid composition and growth, with FDX5 playing a role in electron transfer for lipid desaturation.

Depletion of chloroplast HSP70B causes cell division arrest, proteostasis collapse, impaired VIPP1 oligomer dynamics with aberrant thylakoid structures, and increased light sensitivity.

The paper quantifies protein abundances and stoichiometries in Chlamydomonas reinhardtii during the cell cycle using the total protein approach.

The paper investigates the functional distribution of FDX1 and FDX2 in Chlamydomonas, showing their distinct roles in carbon, nitrogen, and sulfur assimilation through mutant analysis.

This paper designs a nutrient-replete medium for Chromochloris zofingiensis to support fast growth and high triacylglycerol accumulation by optimizing mineral concentrations based on cellular ionome data.

The paper identifies ZNG3 as a Zn chaperone interacting with Cia5 to regulate Zn and CO2 metabolism in Chlamydomonas, with distinct phenotypes in ZNG3 and Cia5 mutants.

This study examines how Chlamydomonas cells adjust to Fe-free conditions, showing rapid transcriptomic changes and slower metabolic shifts.

The paper analyzes two extremophilic algae to understand their iron management strategies, identifying novel iron-uptake proteins and adaptive mechanisms that enable survival in low-iron environments.

The paper optimizes a ChIP-Seq framework for histone modification profiling in Chromochloris zofingiensis and identifies H3K4me3 enrichment at transcription start sites.

The paper shows that reducing histone H3-mediated Cu1+ production restores Fe-S cluster deficiencies in yeast, linking chromatin and mitochondrial function to Fe-S cluster homeostasis.

The paper shows the histone H3-H4 tetramer acts as a copper reductase enzyme, reducing Cu2+ to Cu1+ and affecting cellular processes dependent on reduced copper.