Author

Madhumita Chatterjee

Universitätsklinikum TübingenORCID 0000-0002-3428-4107Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Molecular Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Agnes Bosbach· 3 shared Hubert Schelzig· 3 shared Irena Krüger· 3 shared Kim Jürgen KrottiD· 3 shared Margitta ElversiD· 3 shared Markus Udo WagenhäuseriD· 3 shared Tobias Feige· 3 shared Joscha U MulorziD· 2 shared

Activity by Year

2023-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 43 mutual co-author links

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3 shared papersPathology

Agnes Bosbach

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

PathologyCirculation ResearchPeer-reviewedOct 27, 2025

Crosstalk between thrombospondin-1 and CD36 modulates platelet-RBC interaction limiting thrombosis and abdominal aneurysm formation

Kim Jürgen Krott, Tobias Feige, Agnes Bosbach…·Margitta Elvers

This study identifies a novel CD36-TSP-1 signaling pathway that modulates platelet-RBC interaction, promoting thrombus formation and contributing to abdominal aortic aneurysm pathology.

8 citationstop 4%

humanexperimental assay

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Cell BiologyArteriosclerosis Thrombosis and Vascular BiologyPeer-reviewedAug 29, 2024

Collagen receptor GPVI-mediated platelet activation and pro-coagulant activity aggravates inflammation and aortic wall remodelling in abdominal aortic aneurysm

Tobias Feige, Agnes Bosbach, Kim Jürgen Krott…·Margitta Elvers

This study shows that GPVI-mediated platelet activation worsens AAA by promoting inflammation and aortic wall remodeling, suggesting GPVI as a therapeutic target.

29 citationstop 1%

mousehuman

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Molecular BiologyCardiovascular ResearchPeer-reviewedNov 17, 2023

Crosstalk of platelets with macrophages and fibroblasts aggravates inflammation, aortic wall stiffening, and osteopontin release in abdominal aortic aneurysm

Markus Udo Wagenhäuser, Joscha U Mulorz, Kim Jürgen Krott…·Margitta Elvers

This study shows platelets worsen AAA by promoting inflammation, ECM degradation, and osteopontin release, with platelet depletion reducing aneurysm growth.

82 citationstop 1%

mousehuman

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