Author

Rony Chidiac

Université de SherbrookeORCID 0000-0002-3642-0651Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

Stéphane AngersiD· 2 shared Abdoul-Razak Sango· 1 shared Amitha MuraleedharaniD· 1 shared Benoît VanderperreiD· 1 shared Bora Kiran· 1 shared Carol X.‐Q. CheniD· 1 shared Charlotte Michaud-Tardif· 1 shared Chen Jing· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersNeuroscience

Stéphane AngersORCID

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Author profile →

3 papers

Recent·Most cited

NeurosciencebioRxivPreprintAug 4, 2026

TRIM9 Determines Sex-Specific β-Amyloid/Cellular Prion Protein/mGluR5 Complex Formation and Pathological Signaling in Alzheimer's Disease Mice

Fatemeh Babaei, Fatemeh Panahi Arasi, Tash‐Lynn L. Colson…·Stephen S. G. Ferguson

This study identifies TRIM9 as a sex-specific regulator of the Aβ42/PrPC/mGluR5 complex in male Alzheimer's mice, linking it to pathological signaling and autophagy.

DOI 10.64898/2026.07.30.741783

mouseproteomics

𝕏 PostBluesky

Cell BiologyCommunications BiologyPeer-reviewedOct 6, 2025

A genome-wide CRISPR/Cas9 screen identifies genes that regulate the cellular uptake of α-synuclein fibrils by modulating heparan sulfate proteoglycans

Benoît Vanderperre, Amitha Muraleedharan, Marie‐France Dorion…·Edward A. Fon

This study uses CRISPR/Cas9 screening to identify genes regulating α-synuclein fibril uptake via heparan sulfate proteoglycans, revealing SLC39A9 and C3orf58 as key modulators.

18 citationstop 2%

humangenetic perturbation / CRISPR

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Cell BiologybioRxivPreprintOct 1, 2025

Notch1 induces endothelial plasticity to mediate hyaloid vessel involution

Shaymaa Khazaal, Abdoul-Razak Sango, Megne Ariane Vanessa Megne…·Malika Oubaha

This study identifies Notch1 as a key regulator of hyaloid vessel regression, showing it drives endothelial plasticity and detachment independent of apoptosis.

DOI 10.1101/2025.10.01.679581

mouseexperimental assay

𝕏 PostBluesky