Peter M. Benz

This study shows that Mena and VASP are essential for vascular smooth muscle relaxation, with VASP deletion impairing relaxation responses to acetylcholine and nitric oxide.

The paper shows that reduced H19 lncRNA expression in EVL-deficient mice impairs VEGF expression and bioavailability, leading to compromised angiogenesis in the retina.

This study shows that Ena/VASP proteins are essential for endothelial cell repulsion from ephrin-B2 by mediating actin cytoskeleton collapse and Eph receptor signaling.