Stavros Stavrakis

This paper shows that loss of PFKFB2 in cardiac cells leads to electrophysiological instability, including prolonged repolarization and ventricular tachyarrhythmia, linking glycolytic regulation to heart function.

This study identifies β2 adrenergic receptors as key regulators of neutrophil demargination and recruitment to the ischemic heart after myocardial infarction, showing that β2-AR blockers can improve cardiac remodeling.

This study identifies cardiac myosin autoantibodies associated with reduced ejection fraction and fibrotic transcriptomes in nonrecovered myocarditis patients.

This study shows that loss of cardiac PFKFB2 leads to metabolic, functional, and electrophysiological changes in the heart, including impaired systolic function and altered electrophysiology.