Feng Yue

This study shows that FAM210A deficiency in hepatocytes causes juvenile mitochondrial dysfunction and steatosis, which resolves in adulthood through compensatory mechanisms.

This study shows that inhibiting Pten in adipocytes improves metabolic health by increasing lipid storage, preserving function, and reducing inflammation.

This study shows that autocrine IGF-I is essential for FAP function in muscle homeostasis and regeneration, with loss of IGF-I leading to impaired regeneration and altered FAP properties.

This study identifies chondrolectin as a regulator of satellite cell localization and regenerative function in mice through single-cell RNA-seq and knockout models.

This study identifies NRMLncR, a myocyte-enriched lncRNA, as a regulator of myogenesis in mouse, showing it enhances differentiation through interactions with MyoD/MyoG and CELF1.

The paper uses single nuclei RNA sequencing to analyze muscle from PAD patients and controls, identifying transcriptional changes and cell type alterations associated with the disease.

This study identifies FAM210A as a mediator of mitochondrial-ribosome crosstalk that supports protein synthesis and muscle growth in mice.

This study shows PRMT5 links lipid metabolism to skeletal muscle function by regulating SREBP1a and ATGL through epigenetic mechanisms.

This study shows that PTEN inhibition reduces muscle degeneration and improves function in mdx mice, a model of Duchenne Muscular Dystrophy.