Andrew S. Wagner

This paper establishes a zebrafish burn wound model to study fungal colonization and host immune responses, finding that macrophages and neutrophils control fungal infection with some redundancy.

This study shows that GATA1-deficient iPSC-derived neutrophils have enhanced antifungal activity against Aspergillus fumigatus, mediated by CD18 integrin.

The paper shows isotonic medium treatment reduces epithelial damage spread and microbial colonization in burn wounds in larval zebrafish and human skin.

This study shows that human neutrophils shift nutrient preference from glycogen to glucose upon activation, with function-specific metabolic dependencies.