Maximillian E. Denys

This study shows that early-life exposure to PBDEs in mice leads to autism-like behaviors and changes in prosocial neuropeptide expression.

This study shows that Limosilactobacillus reuteri supplementation mitigates PBDE-induced ASD-like social and neuroendocrine deficits in mice by modulating the gut microbiome-thyroid-oxytocin axis.

This study shows maternal thyroid supplementation prevents PBDE-induced social behavior deficits and hypothalamic oxytocin depletion in mice.

This study shows that deleting the VPAC2R gene in female mice increases basal glycemia and impairs insulin response during metabolic stress, while also enhancing hyperglycemia during psychogenic stress.

This study shows that exposure to PBDE mixture DE-71 during pregnancy and lactation in female mice leads to autism-relevant behavioral and neurochemical changes in offspring.