Author

Mélèze Hocini

Electrophysiology and Heart Modeling InstituteORCID 0000-0002-7957-8766Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:electronic health records· 1 Assay:imaging· 1

Frequent Co-Authors

Michel Haı̈ssaguerreiD· 2 shared Agnès TessieriD· 1 shared Akinola Alafiatayo· 1 shared Alice BoissardiD· 1 shared Bastien CimarostiiD· 1 shared Benjamin LauzieriD· 1 shared Bruno D. M. Y. StuyversiD· 1 shared Bruno QuessoniD· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersPathology

Michel Haı̈ssaguerreORCID

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2 papers

Recent·Most cited

PathologybioRxivPreprintAug 11, 2026

Desmin p.R406W mutation is associated with arrhythmias through structural and electrophysiological remodeling

Michelle Geryk, Thomas Stervinou, Martin Bouaud…·Flavien Charpentier

This study investigates the pathogenic effects of the DES p.R406W mutation, showing it causes electrical and structural heart remodeling linked to arrhythmias and sudden cardiac death.

DOI 10.64898/2026.08.05.742729

humanmouse

𝕏 PostBluesky

Cell BiologybioRxivPreprintOct 23, 2023

Increased spontaneous Ca2+ activity in Cardiac Purkinje cells after myocardial infarction; A consequence of a dramatic shift of SERCA isoforms as potential adaptation to acute ischemia?

Bruno D. M. Y. Stuyvers, Penelope A. Boyden, Ruhul Amin…·Touati Benoukraf

This study shows that increased spontaneous Ca2+ activity in cardiac Purkinje cells after myocardial infarction is due to a shift in SERCA2 isoforms, specifically increased SERCA2b and reduced SERCA2a.

DOI 10.1101/2023.10.23.563678

humanmouse

𝕏 PostBluesky