Author

David J. Weber

University of Maryland, BaltimoreORCID 0000-0002-8824-1110Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Biophysics· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

Raquel Godoy‐RuiziD· 2 shared Alagie JasseyiD· 1 shared Alejandro X. Spittel· 1 shared Amédée des GeorgesiD· 1 shared Bimal Paudel· 1 shared Dinendra L. AbeyawardhaneiD· 1 shared Edwin V. PozharskiiD· 1 shared I-Ting Cheng· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersMicrobiology

Raquel Godoy‐RuizORCID

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2 papers

Recent·Most cited

MicrobiologyScience AdvancesPeer-reviewedApr 22, 2026

Mitophagosomes induced during EV-D68 infection promote viral nonlytic release

Alagie Jassey, Bimal Paudel, Michael A. Wagner…·William T. Jackson

This study shows EV-D68 induces mitophagy to facilitate nonlytic viral release, distinct from its role in supporting viral replication.

2 citations

humanexperimental assay

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BiophysicsbioRxivPreprintAug 18, 2023

Membrane binding and pore formation is Ca2+-dependent for the Clostridioides difficile binary toxin

Dinendra L. Abeyawardhane, Spiridon E. Sevdalis, Kaylin A. Adipietro…·David J. Weber

This study shows that Ca2+ dissociation from CDTb's RBD1 triggers pore formation in endosomal membranes, differing from other binary toxins that rely on pH changes.

DOI 10.1101/2023.08.18.553786

experimental assay

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