Ronni A. G. da Silva

This study shows E. faecalis biofilms suppress neutrophil defenses by producing lactic acid and high bacterial density.

This paper shows that E. faecalis uses lactic acid to suppress macrophage activation, enabling persistent and polymicrobial wound infections.

This study identifies the Fsr quorum sensing system in Enterococcus faecalis as a negative regulator of biofilm growth and inflammation in infective endocarditis.

This study shows Enterococcus faecalis persists and replicates intracellularly within neutrophils, prolonging infection and inflammation in chronic wounds.

This paper shows bosutinib enhances macrophage phagocytosis and bacterial killing, reducing infection in mice.