Author

Iddo Z. Ben‐Dov

Hadassah Medical CenterORCID 0000-0002-9180-3604Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Cancer Biology· 1 Developmental Biology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1 Organism:rat· 1

Frequent Co-Authors

Alia HassaniD· 2 shared Rachel KilaviD· 2 shared Tally Naveh‐ManyiD· 2 shared Barbara Del CastelloiD· 1 shared Harel ReinusiD· 1 shared Justin Silver· 1 shared Morris NechamaiD· 1 shared Nancy Ruth ManleyiD· 1 shared

Activity by Year

2022-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersDevelopmental Biology

Alia HassanORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Cancer BiologybioRxivPreprintSep 26, 2024

Characterization of alternative mRNA splicing associated with tumor thrombus in clear-cell renal cell carcinoma

Zeev Cohen, Harel Reinus, Iddo Z. Ben‐Dov, Tomer Kalisky C

The paper analyzes RNAseq data to identify alternative splicing patterns associated with tumor thrombus formation in clear-cell renal cell carcinoma.

DOI 10.1101/2024.09.26.615158

humanbulk RNA-seq

𝕏 PostBluesky

Developmental BiologyJournal of the American Society of NephrologyPeer-reviewedMay 31, 2024

The essential roles of Dicer-mediated mTORC1 signaling in parathyroid gland integrity and function: Insights from genetic mouse models and clinical data.

Alia Hassan, Nareman Khalaily, Rachel Kilav…·Tally Naveh‐Many

This study investigates the role of Dicer-mediated mTORC1 signaling in parathyroid function using genetic mouse models and clinical data, showing its critical role in maintaining gland integrity and response to CKD.

5 citations

mousestatistical model

𝕏 PostBluesky

Molecular BiologyJournal of the American Society of NephrologyPeer-reviewedAug 12, 2022

Kidney failure alters parathyroid Pin1 phosphorylation and parathyroid hormone mRNA binding proteins leading to secondary hyperparathyroidism

Alia Hassan, Yael E. Pollak, Rachel Kilav…·Tally Naveh‐Many

The paper identifies a pathway where kidney failure induces parathyroid Pin1 phosphorylation, disrupting KSRP activity and increasing PTH mRNA stability, leading to secondary hyperparathyroidism.

8 citations

𝕏 PostBluesky