James G. Xenakis

This paper introduces a deep neural network-based two-part model for semi-continuous data and demonstrates its improved performance over existing methods.

The paper identifies genetic variants associated with collateral blood vessel abundance in mice, linking them to potential human disease mechanisms.

The paper identifies MBD1 as a regulator of homeostatic IgG1 levels and marginal zone B cell differentiation through a genetic screen in Collaborative Cross mice.

This study identifies genetic factors influencing antipsychotic-induced adverse reactions in mice using RIX lines, finding strong strain and treatment interactions for behavioral phenotypes.