Author

W. C. K. Wang

University of British ColumbiaORCID 0000-0002-9485-583XUnclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

W. C. K. Wang1 paper

Biology Disciplines

Click to filter author papers

Cancer Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:development & morphogenesis· 1 Approach:experimental assay· 1 Assay:imaging· 1 Organism:mouse· 1

Frequent Co-Authors

David F. SchaefferiD· 1 shared Gloria H. SuiD· 1 shared Ian S. Wood· 1 shared Janel L. KoppiD· 1 shared Kiyoshi SaekiiD· 1 shared Shilpa PatiliD· 1 shared Yanping SuniD· 1 shared

Activity by Year

2024-2024 · peak 1/yr

Co-Author Collaboration Network

7 top collaborators · 21 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCancer Biology

David F. SchaefferORCID

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

Cancer BiologyCellular and Molecular Gastroenterology and HepatologyPeer-reviewedJan 1, 2024

Acvr1b loss increases formation of pancreatic precancerous lesions from acinar and ductal cells of origin

Kiyoshi Saeki, Ian S. Wood, W. C. K. Wang…·Janel L. Kopp

This study shows that Acvr1b loss in combination with Kras mutation promotes precancerous lesions from both acinar and ductal cells in the pancreas, but the IPMN-like phenotype differs when mutations occur in all cells.

5 citations

𝕏 PostBluesky