Author

Vaya Chen

ORCID 0000-0002-9824-4809Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Vaya Chen1 paper · Versiti Blood Center of Wisconsin

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Mirza Ahmar BegiD· 2 shared Weiguo CuiiD· 2 shared Yaxin WangiD· 2 shared Yiliang CheniD· 2 shared A Wayne Orr· 1 shared Ankan GuptaiD· 1 shared Ashley Kaye Brown· 1 shared Brian C. SmithiD· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersImmunology

Mirza Ahmar BegORCID

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2 papers

Recent·Most cited

ImmunologyArteriosclerosis Thrombosis and Vascular BiologyPeer-reviewedAug 6, 2026

Macrophage PIM1 Drives Atherosclerosis by Enhancing Foam Cell Formation Via CD36

Mirza Ahmar Beg, Quoc Quang Luu, Wenjing Chen…·Yiliang Chen

This study shows that PIM1 in macrophages enhances CD36 expression and function, promoting foam cell formation and atherosclerosis, and that targeting this pathway could reduce plaque progression.

DOI 10.1161/atvbaha.126.324938

mousebulk RNA-seq

𝕏 PostBluesky

ImmunologybioRxivPreprintSep 7, 2023

Oxidized LDL regulates efferocytosis through the CD36-PKM2-mtROS pathway

Jue Zhang, Jackie Chang, Vaya Chen…·Yiliang Chen

This study identifies the CD36-PKM2-mtROS pathway as a mechanism by which oxidized LDL disrupts efferocytosis, contributing to atherosclerosis progression.

DOI 10.1101/2023.09.07.556574

mouseproteomics

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