Cristina Colomer‐Winter

This study identifies a two-component system signaling hub that controls membrane remodeling in Enterococcus faecalis in response to daptomycin, linking lipid changes to antibiotic resistance.

This study uses human colonic organoids to reveal how Enterococcus faecalis adapts to intestinal mucus, identifying key fitness determinants and colonization strategies not observed in vitro or in vivo.

This study identifies the Fsr quorum sensing system in Enterococcus faecalis as a negative regulator of biofilm growth and inflammation in infective endocarditis.

This paper shows that HtrA clears off-pathway pili in E. faecalis, leading to membrane stress and antibiotic susceptibility.