Author

Patricia Prada-Dacasa

Universitat Autònoma de BarcelonaORCID 0000-0003-0689-9072Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Neuroscience· 3

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:differential expression· 2 Assay:ATAC-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

Albert QuintanaiD· 3 shared Elisenda SanziD· 3 shared Alejandro GellaiD· 2 shared Melania Gonzalez-TorresiD· 2 shared Abel Eraso‐PichotiD· 1 shared Alessandro PrigioneiD· 1 shared Alex Gella· 1 shared Andrea UrpiiD· 1 shared

Activity by Year

2019-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersNeuroscience

Albert QuintanaORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

NeurosciencebioRxivPreprintNov 12, 2026

PKR drives neuropathology in a mouse model of Leigh syndrome

Melania Gonzalez-Torres, Patrizia Bianchi, Marcos Blanco-Ramos…·Albert Quintana

The paper identifies PKR as a critical mediator of mitochondrial neuropathology in a mouse model of Leigh syndrome, showing its role in neuroinflammation and disease progression.

DOI 10.64898/2026.11.10.765677

mouseATAC-seq

𝕏 PostBluesky

NeuroscienceFrontiers in Cell and Developmental BiologyPeer-reviewedJul 28, 2020

Mitochondrial Proteome of Affected Neurons in a Mouse Model of Leigh Syndrome

Alejandro Gella, Patricia Prada-Dacasa, Montserrat Carrascal…·Albert Quintana

The paper uses a novel viral tool to isolate mitochondria from affected neurons in a mouse model of Leigh Syndrome, revealing proteomic and acetylomic changes associated with mitochondrial dysfunction.

16 citations

mouseproteomics

𝕏 PostBluesky

NeuroscienceeLifePeer-reviewedAug 12, 2019

Defined neuronal populations drive fatal phenotype in Leigh Syndrome

Irene Bolea, Alejandro Gella, Elisenda Sanz…·Albert Quintana

The paper identifies two genetically-defined neuronal populations in Ndufs4KO mice that drive distinct pathological features of Leigh Syndrome.

57 citations

mouseexperimental assay

𝕏 PostBluesky