Dominique C. Bergmann

The paper identifies a polarity circuit in guard cells that determines stomatal pore position through MUS and OPL2 interactions, revealing a mechanism for coordinated cellular asymmetry.

The paper shows that the ACTL domain in bHLH transcription factors determines partner specificity by affecting dimerization and function in vivo.

The paper identifies late stomatal lineage targets of SPCH and FAMA, showing their roles in controlling guard cell division and expansion through temporal regulation.

The paper identifies developmental rules governing stomatal patterning in Arabidopsis by analyzing variation across accessions and environmental conditions.

The paper identifies SlSPCH variants affecting stomatal development in tomato in response to environmental cues using CRISPR/Cas9 and live-cell reporters.

The paper investigates SPCH gene duplications in grasses and shows how paralog-specific divergence in response to environmental changes affects stomatal development.

The paper investigates how cell size influences division potential in Arabidopsis stomatal lineage ground cells through multi-scale mechanisms.

The paper identifies polarity domains in Arabidopsis stomatal lineage cells and their associated proteins using proteomics and structural modeling, revealing their role in cell division and pore formation.

The paper shows that a cell size threshold triggers commitment to stomatal fate in Arabidopsis by integrating computational simulations and experimental validation.

The paper identifies a stem-cell-ubiquitous gene, TCX2, that coordinates division across different stem cell types in Arabidopsis by regulating stem cell-specific networks.

The paper identifies the plant-specific BRX family as partners of BASL in establishing cell fate asymmetry in Arabidopsis stomatal lineage through polar localization and mutual dependencies.

This study identifies SOL1 and SOL2 as regulators of fate transitions and cell divisions in the Arabidopsis stomatal lineage, showing their role in cell cycle-dependent regulation and interaction with TSO1.