Author

Kwasi Ankomah

Komfo Anokye Teaching HospitalORCID 0000-0003-1290-8406Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:drug response / drug discovery· 2 Organism:human· 2 Assay:organoid / primary culture· 2 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

Baffour AwuahiD· 2 shared Brian StonakeriD· 2 shared Ernest Baawuah Osei-BonsuiD· 2 shared Esther ChengiD· 2 shared Florencia P. Madorsky RowdoiD· 2 shared Ishmael KyeiiD· 2 shared John NguyeniD· 2 shared Kofi K. GyaniD· 2 shared

Activity by Year

2023-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersCancer Biology

Baffour AwuahORCID

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2 papers

Recent·Most cited

Cancer BiologyCancer ResearchPeer-reviewedFeb 1, 2025

Kinome focused CRISPR-Cas9 screens in African ancestry patient-derived breast cancer organoids identifies essential kinases and synergy of EGFR and FGFR1 inhibition.

Florencia P. Madorsky Rowdo, Rachel N. Martini, Sarah E. Ackermann…·M. Laura Martín

The paper uses CRISPR-Cas9 screens in breast cancer organoids from African ancestry patients to identify essential kinases and show synergy between EGFR and FGFR1 inhibitors.

23 citationstop 3%

humanorganoid / primary culture

𝕏 PostBluesky

Cancer BiologybioRxivJun 22, 2023

Patient-derived tumor organoids with p53 mutations, and not wild-type p53, are sensitive to synergistic combination PARP inhibitor treatment

Florencia P. Madorsky Rowdo, Gu Xiao, Galina F. Khramtsova…·Jill Bargonetti

This study shows that tumor organoids with mutant p53 are more sensitive to the combination of PARP inhibitor and temozolomide than those with wild-type p53.

DOI 10.1016/j.canlet.2024.216608

humanorganoid / primary culture

𝕏 PostBluesky