Matheus Batista Heitor Carneiro

The paper shows that pre-existing Th1 immunity is overcome by monocyte-derived macrophages that create a pathogen niche, and that checkpoint blockade or depletion of these cells improves parasite control.

This study shows impaired lymphatic function contributes to non-healing cutaneous leishmaniasis caused by L. amazonensis and demonstrates that targeting lymphatics can reduce inflammation.

This study identifies APOL7C as a pore-forming protein that facilitates phagosomal rupture and antigen cross-presentation by cDC1s.