Samantha Ottinger

This study identifies immune signatures in mice and human urine that predict preterm birth risk following UTI, highlighting cytokines like IL-10 and IL-15 as potential therapeutic targets.

This study shows that type 2 diabetes increases susceptibility to vaginal group B Streptococcus colonization through impaired cytokine responses, not via glucose availability or microbiome changes.

This study develops human bladder organoids to model UTIs and phage therapy, showing phage reduces bacterial loads and inflammation during infection.

This study shows that gestational diabetes increases GBS perinatal infection by altering maternal immunity and vaginal microbiota, using a murine model.