Johnathan A. Napier

The paper identifies mutations in TREX-2 complex components SAC3B and THP1 in Arabidopsis that cause misexpression of the KFB39 promoter, suggesting a potential regulatory link between the complex and KFB39 expression.

The paper identifies DGK1 and DGK2 as forming a module with SYT1 at ER-PM contact sites in Arabidopsis, enhancing PI cycle efficiency through DAG transport and phosphorylation during stress.

The paper shows that synaptotagmins maintain diacylglycerol homeostasis at ER-PM contact sites during abiotic stress by preventing its detrimental accumulation at the plasma membrane.