Author

Bezawit T. Danna

University of California, Los AngelesORCID 0000-0003-4114-1275Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Biochemistry· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:metabolomics· 3 Research Focus:metabolism & bioenergetics· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Ajit S. DivakaruniiD· 3 shared Linsey StilesiD· 2 shared Orian S. ShirihaiiD· 2 shared Ahmad ShamiiD· 1 shared Alexander R. Strofs· 1 shared Ambre M. Bertholet· 1 shared Amy Rios· 1 shared Andréa B BalliD· 1 shared

Activity by Year

2024-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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3 shared papersBiochemistry

Ajit S. DivakaruniORCID

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3 papers

Recent·Most cited

Cell BiologybioRxivPreprintJul 28, 2025

Saturated lipid stress attenuates mitochondrial genome synthesis in human cells

Casadora Boone, Sophie Judge, Ahmad Shami…·Samantha C. Lewis

The paper shows that saturated lipid stress reduces mtDNA synthesis in human cells by disrupting ER-mitochondria contact sites and fatty acid trafficking, which is reversed by monounsaturated fatty acids.

1 citation

humanbulk RNA-seq

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BiochemistrybioRxivPreprintApr 30, 2025

13C tracing in synaptosomes reveals that SGLT2 inhibition with dapagliflozin prevents metabolic deficits in the 5X-FAD model of Alzheimer's Disease

Marisa Mekkittikul, Cansheng Zhu, Bezawit T. Danna…·Ajit S. Divakaruni

1 citation

mousemetabolomics

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BiochemistryJournal of Biological ChemistryPeer-reviewedJan 30, 2024

The BCKDK inhibitor BT2 is a chemical uncoupler that lowers mitochondrial ROS production and de novo lipogenesis

Aracely Acevedo, Anthony E. Jones, Bezawit T. Danna…·Ajit S. Divakaruni

The paper shows BT2 acts as a mitochondrial uncoupler, reducing ROS and lipogenesis, which may explain its therapeutic benefits in metabolic diseases.

32 citations

mousemetabolomics

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