Steven H. Spoel

This study shows that HECT-type ligases UPL3/4 promote the degradation of other E3 ligases like PUB22, thereby regulating immune-related substrate stability and plant immunity.

This study shows that GSNOR-dependent nitric oxide homeostasis aids recovery from repeated climate stress in Arabidopsis across generations, while its impairment leads to persistent stress responses.

This study identifies ubiquitin recognition as the mechanism enabling mutual potentiation between cell-surface and intracellular receptors in plant immunity.

This study identifies TGA transcription factors, particularly PAN, as nitric oxide sensors in the root stem cell niche, revealing their role in modulating gene expression and stem cell maintenance through S-nitrosylation.

This study shows that overexpression of the clock gene TOC1 suppresses PAMP-triggered immune responses in Arabidopsis, increasing susceptibility to Pseudomonas syringae.

The paper shows mobile signals like NHP enhance SA-mediated plant immunity through WRKY38/62 transcription factors, while AzA dampens it.

This study shows that salicylic acid signaling and the circadian clock interact to enhance immune responses in Arabidopsis against bacterial pathogens.

This study shows coronatine toxin suppresses plant immunity by disrupting glutathione and nitric oxide redox signaling, independent of jasmonic acid pathways.