Author

Thomas A. Kufer

University of HohenheimORCID 0000-0003-4563-0412Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

T. A. Kufer1 paper

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Organism:mouse· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:protein folding & dynamics· 1

Frequent Co-Authors

A QuenumiD· 1 shared A. De Paoli· 1 shared A. Mansell· 1 shared Adrian Beck· 1 shared Akhil ShuklaiD· 1 shared Anny Armas Cayarga· 1 shared B. A. Croker· 1 shared C. C. Allison· 1 shared

Activity by Year

2019-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 12 mutual co-author links

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1 shared paperImmunology

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3 papers

Recent·Most cited

ImmunologybioRxivPreprintDec 23, 2025

NLRC5 expression in tumor cells is critical to activate adaptive and innate antitumor immune responses

Akhil Shukla, A Quenum, Jean‐François Lucier…·Subburaj Ilangumaran

This study shows that NLRC5 expression in tumor cells enhances MHC-I and promotes antitumor immunity by activating immune cells and shaping the tumor microenvironment.

1 citation

mouseproteomics

𝕏 PostBluesky

ImmunologybioRxivPreprintNov 27, 2025

C-terminal lysine residues localise NLRP10 at lipid compartments and govern NLRP10 oligomer formation

Timo-Daniel Voß, Christoph Winterberg, Adrian Beck…·Thomas A. Kufer

This study identifies the C-terminal lysine residues of NLRP10 as critical for its localization to lipid compartments and oligomer formation, highlighting unique activation mechanisms compared to other NLRPs.

DOI 10.1101/2025.11.27.690698

𝕏 PostBluesky

ImmunologybioRxivPreprintMar 24, 2019

NOD1 mediates non-canonical inflammasome processing of interleukin-18 in epithelial cells to Helicobacter pylori infection.

L. S. Tran, L. Ying, K. D’Costa…·R. L. Ferrero

This study identifies NOD1 as a key regulator of IL-18 processing in epithelial cells during Helicobacter pylori infection, independent of canonical inflammasome proteins.

DOI 10.1101/587212

humanexperimental assay

𝕏 PostBluesky