Kevin R. Foster

The paper investigates bacterial warfare by analyzing toxin production in Escherichia coli, finding that localized toxin bursts are more effective than uniform production in structured populations.

This paper develops a modular antimicrobial platform using engineered SimCells and mini-SimCells to selectively eliminate E. coli with high specificity and efficiency.

The paper investigates why bacteria carry multiple similar weapons, finding that they are specialised for different environmental conditions.

This study links bacterial warfare via bacteriocins to virulence and antimicrobial resistance in E. coli, showing their association with pathogenic strains and horizontal gene transfer.

This study models and tests how horizontal gene transfer affects bacterial warfare, finding that gene transfer can benefit weapon genes but not the bacteria carrying them under certain conditions.

The paper uses time-resolved single-cell imaging to study the Escherichia coli oxidative stress response, revealing simple regulatory logic in gene expression dynamics.

This study shows that Pseudomonas aeruginosa uses spatial sensing, not temporal sensing, to detect chemical gradients during surface-based chemotaxis.

The paper shows that bacterial motility enhances contact-based toxin delivery by increasing inter-strain contact and target switching, improving CDI effectiveness up to 10,000-fold.