Author

Martina Milani

University of Rome Tor VergataORCID 0000-0003-4889-6988Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Martina Milani1 paper · European Institute of Oncology

Biology Disciplines

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Neuroscience· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:host-pathogen & immune response· 1 Research Focus:metabolism & bioenergetics· 1 Assay:metabolomics· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Ilaria Della ValleiD· 2 shared Nadia D’AmbrosiiD· 2 shared Savina ApolloniiD· 2 shared Andrea Ninni· 1 shared Annalucia SerafinoiD· 1 shared Cinzia VolontéiD· 1 shared Daniele Lettieri‐BarbatoiD· 1 shared Erica D’Avorio· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersNeuroscience

Ilaria Della ValleORCID

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2 papers

Recent·Most cited

NeuroscienceCell Death and DiseasePeer-reviewedMar 29, 2025

Cytoplasmic accumulation of a splice variant of hnRNP A2/B1 contributes to FUS-associated toxicity in a mouse model of ALS

Simona Rossi, Martina Milani, Ilaria Della Valle…·Mauro Cozzolino

This study shows that FUS-induced splicing changes lead to cytoplasmic accumulation of hnRNP A2b/B1 splice variants, promoting stress granule formation and motor neuron toxicity in a mouse model of ALS.

6 citations

mouseexperimental assay

𝕏 PostBluesky

PhysiologyLife Science AlliancePeer-reviewedApr 17, 2024

Frataxin Deficiency Drives a Shift from Mitochondrial Metabolism to Glucose Catabolism, Triggering an Inflammatory Phenotype in Microglia

Francesca Sciarretta, Fabio Zaccaria, Andrea Ninni…·Daniele Lettieri‐Barbato

This study shows that frataxin deficiency in microglia shifts metabolism to glucose catabolism, causing inflammation, which is mitigated by butyrate treatment.

15 citations

𝕏 PostBluesky