Author

Jonathan Kölschbach

Max Planck Institute for Biology of AgeingORCID 0009-0000-5191-4732Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Genetics· 1

Signature Assays, Organisms & Methods

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Organism:C. elegans· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Adam AntebiiD· 2 shared Anna Löhrke· 1 shared Birgit Gerisch· 1 shared Christian Latza· 1 shared Chun KewiD· 1 shared Emily Baum· 1 shared Hormos Salimi DafsariiD· 1 shared Ivan ĐikićiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersGenetics

Adam AntebiORCID

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2 papers

Recent·Most cited

GeneticsbioRxivPreprintSep 29, 2025

Vitamin B12 alleviates Verheij syndrome-like defects via phospholipid remodeling in a C. elegans PUF60 spliceosomopathy model

Wenming Huang, Jonathan Kölschbach, Hormos Salimi Dafsari…·Adam Antebi

This study identifies vitamin B12 as a suppressor of Verheij syndrome-like defects in a C. elegans PUF60 model by restoring metabolic balance and splicing.

DOI 10.1101/2025.09.29.678827

C. elegansbulk RNA-seq

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Cell BiologyNature AgingPeer-reviewedJun 30, 2025

A primordial TFEB-TGFβ signaling axis systemically regulates diapause and stem cell longevity

Tim J. Nonninger, Jennifer Mak, Birgit Gerisch…·Adam Antebi

The paper identifies a TFEB-TGFβ signaling axis that regulates diapause and stem cell longevity across species by coordinating nutrient availability with growth signals.

25 citationstop 4%

C. elegansgenetic perturbation / CRISPR

𝕏 PostBluesky