Author

Erin A. Fowler

The Ohio State UniversityORCID 0009-0001-7746-8985Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell signaling & trafficking· 2 Organism:human· 2 Organism:mouse· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Fernanda O. NovaisiD· 3 shared Aditi Varkey· 1 shared Allison Yan· 1 shared Camila Farias Amorim· 1 shared Camila I. de Oliveira· 1 shared Emily D. S. Hales· 1 shared Faaiza Saif· 1 shared Gary D. Wu· 1 shared

Activity by Year

2023-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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3 shared papersImmunology

Fernanda O. NovaisORCID

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3 papers

Recent·Most cited

ImmunologybioRxivPreprintAug 4, 2026

Hypoxia induces cytotoxicity and suppresses cytokine production by CD8⁺ T cells in cutaneous leishmaniasis.

Erin A. Fowler, Olivia L. Schneider, Faaiza Saif, Fernanda O. Novais

This study shows hypoxia in leishmaniasis lesions reprograms CD8⁺ T cells to become cytotoxic while suppressing protective cytokine production.

DOI 10.64898/2026.07.30.741849

humanproteomics

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ImmunologybioRxivPreprintJun 9, 2026

Acriflavine treatment attenuates inflammatory pathology in cutaneous leishmaniasis independently of parasite control

Erin A. Fowler, Fernanda O. Novais

This study shows acriflavine reduces cutaneous leishmaniasis lesion size in mice without affecting parasite burden, by suppressing dendritic cell activation and IFN-gamma-driven immunopathology.

1 citation

mouseexperimental assay

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ImmunologybioRxivPreprintOct 18, 2023

Pathogenic CD8 T cell responses are driven by neutrophil-mediated hypoxia in cutaneous leishmaniasis.

Erin A. Fowler, Camila Farias Amorim, Klauss Mostacada…·Fernanda O. Novais

The paper shows that hypoxia, driven by neutrophils, promotes cytolytic CD8 T cell function in cutaneous leishmaniasis lesions, correlating with disease severity.

DOI 10.1101/2023.10.18.562926

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