Keena S. Thomas

This paper develops a microphysiologic model of the cervical epithelium that replicates microbial, immunologic, and pathogenic features of STIs caused by Chlamydia and Neisseria.

This study investigates cross-species immune responses induced by meningococcal OMV vaccines and their association with protection against N. gonorrhoeae.

This paper shows that Neisseria gonorrhoeae uses sialic acid from the host to modify its surface, which helps it avoid neutrophil activation without needing the complement system.