Hunter J. Hill

This study maps dicentric chromosome breakage hotspots in Drosophila and identifies structural complexity, replication-transcription conflict, and transposon involvement as key factors.

The paper shows Wolbachia-induced chromatin modifications suppress position-effect variegation in Drosophila, persisting into adulthood and not fully reversed by CifA.

The paper identifies five causes of anaphase bridges formed by ring chromosomes in Drosophila melanogaster, including sister chromatid catenation and homolog capture.

The paper studies dicentric chromosome breakage in Drosophila melanogaster and finds that breakage hotspots are not conserved and are influenced by pericentric heterochromatin and centromere origin.