Author

Natalia Athanasiadi

The Francis Crick InstituteORCID 0009-0006-4453-0611Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Organism:human· 3 Research Focus:differential expression· 2 Organism:bacteria (other)· 1 Assay:bulk RNA-seq· 1 Research Focus:drug response / drug discovery· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Maximiliano Gabriel GutierreziD· 3 shared Chak Hon LukiD· 2 shared Enrica PellegrinoiD· 2 shared Angela RodgersiD· 1 shared Antony FearnsiD· 1 shared Claudio BussiiD· 1 shared Gabriel L. Conway· 1 shared Huw R. MorrisiD· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersCell Biology

Maximiliano Gabriel GutierrezORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

BioengineeringScience AdvancesPeer-reviewedJan 1, 2026

✦ In-depth digest

Autologous human iPSC-derived Alveolus-on-Chip reveals early pathological events of M. tuberculosis infection

Chak Hon Luk, Gabriel L. Conway, Kim Jee Goh…·Maximiliano Gabriel Gutierrez

A single-donor iPSC-derived alveolar chip with epithelial, endothelial and macrophage cells models early Mycobacterium tuberculosis infection and shows ATG14-deficient macrophages die more without extra bacterial replication.

10 citationstop 1%

humanbacteria (other)

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Cell BiologymBioPeer-reviewedJul 10, 2024

Physiologic media renders human iPSC-derived macrophages permissive for M. tuberculosis by rewiring organelle function and metabolism

Claudio Bussi, Rachel Lai, Natalia Athanasiadi, Maximiliano Gabriel Gutierrez

This study shows that using a human plasma-like medium enhances M. tuberculosis replication in iPSC-derived macrophages by altering lipid metabolism and organelle function.

15 citationstop 4%

humanbulk RNA-seq

𝕏 PostBluesky

Cell BiologybioRxivPreprintSep 27, 2023

LRRK2 kinase dependent and independent function on endolysosomal repair promotes macrophage cell death

Rebecca Morrison, Simeon R. Mihaylov, Chak Hon Luk…·Maximiliano Gabriel Gutierrez

This study identifies LRRK2-G2019S-dependent lysosomal membrane repair defects in macrophages leading to increased cell death.

1 citation

humanmouse

𝕏 PostBluesky