Author

A. Alvarez-Prado

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:clustering· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

A. de Molina· 1 shared A. Durandy· 1 shared A. R. Ramiro· 1 shared B. Grimbacher· 1 shared C. Klemann· 1 shared C. Plass· 1 shared C. Speckmann· 1 shared D. Weichenhan· 1 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCancer Biology

A. de Molina

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Interplay between UNG and AID governs intratumoral heterogeneity in mature B cell lymphoma

A. R. Ramiro, P. Delgado, A. Alvarez-Prado, E. Marina-Zarate, I. Sernandez, S. M. Mur, J. de la Barrera, F. Sanchez-Cabo, M. Canamero, A. de Molina, L. Belver, V. G. de Yebenes

The paper shows that UNG limits AID-induced intratumoral heterogeneity in B cell lymphoma by suppressing SHM and oncogenic variants when AID is overexpressed.

bioRxiv · Jun 29, 2020Cancer Biology

Activation-induced deaminase is critical for the establishment of DNA methylation patterns prior to the germinal center reaction

F. Catala-Moll, D. Weichenhan, P. Lutsik, A. Alvarez-Prado, J. Rodriguez-Ubreva, C. Klemann, C. Speckmann, H. Abolhassani, M. Martinez-Gallo, R. Dieli-Crimi, P. Soler-Palacin, S. Kracker, L. Hammarstrom, A. Durandy, B. Grimbacher, , E. Ballestar

This study investigates AID's role in DNA methylation defects in HIGM2 B cells, finding that AID is not directly involved in active demethylation but TET enzymes are, and that methylation defects are linked to premature BCR activation.

bioRxiv · Aug 16, 2019Immunology