Author

A. Batut

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 2 Bioengineering· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

A. Montagner· 2 shared A. Polizzi· 2 shared C. Postic· 2 shared H. Guillou· 2 shared A. Dortignac· 1 shared A. Fougerat· 1 shared A. Yart· 1 shared B. Boudra· 1 shared

Activity by Year

2019-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersPathology

A. Montagner

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Author profile →

3 papers

Recent·Most cited

Deposition chamber technology as building blocks for a standardized brain-on-chip framework

B. G. C. Maisonneuve, L. Libralesso, L. Miny, A. Batut, J. Rontard, M. Gleyzes, B. Boudra, J. Vieira, D. Debis, F. Larramendy, V. Jost, T. Honegger

This paper introduces a microfluidic platform for creating multi-nodal neuronal circuits to better model brain connectomes for studying neurodegenerative diseases.

bioRxiv · Jun 21, 2021Bioengineering

Nuclear HMGB1 protects from non-alcoholic fatty liver diseases through negative regulation of liver X receptor

J. Personnaz, E. Piccolo, A. Dortignac, J. S. Iacovoni, J. Mariette, A. Polizzi, A. Batut, S. Deleruyelle, R. Paccoud, E. Moreau, F. Martins, T. Clouaire, F. Benhamed, A. Montagner, W. A. Wahli, , J.-P. Pradere

This study shows that nuclear HMGB1 suppresses liver lipogenesis in NAFLD by inhibiting LXR activity, preventing steatosis and insulin resistance.

bioRxiv · Feb 17, 2021 · PreprintPathology

Hepatocyte-specific deletion of Pparα promotes NASH in the context of obesity

M. Regnier, A. Polizzi, S. Smati, C. Lukowicz, A. Fougerat, Y. Lippi, E. Fouche, F. Lasserre, C. Naylies, C. Betoulieres, V. Barquissau, E. Mouisel, J. Bertrand-Michel, A. Batut, T. Al Saati, , A. Montagner

This study shows that deleting Ppar in hepatocytes promotes NASH in obese mice without glucose intolerance, highlighting the role of extra-hepatocyte PPAR in metabolic responses to HFD.

bioRxiv · Jan 18, 2019 · PreprintPathology