Author

E. Fouche

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

A. Fougerat· 2 shared A. Montagner· 2 shared A. Polizzi· 2 shared C. Canlet· 2 shared C. Lukowicz· 2 shared C. Naylies· 2 shared C. Postic· 2 shared F. Lasserre· 2 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersCell Biology

A. Fougerat

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

p110alpha-dependent hepatocyte signaling is critical for liver gene expression and its rewiring in MASLD

M. Regnier, A. Polizzi, T. Fougeray, A. Fougerat, P. Perrier, K. Anderson, Y. Lippi, S. Smati, C. Lukowicz, F. Lasserre, E. Fouche, M. Huillet, C. Rives, B. Tramunt, C. Naylies, , H. Guillou

This study investigates the role of p110alpha in hepatocyte signaling, showing its critical impact on liver gene expression and its rewiring in MASLD.

bioRxiv · Jan 9, 2024 · PreprintCell Biology

Reduction, removal or replacement of sodium nitrite in a model of cured and cooked meat: a joint evaluation of consequences on microbiological issues in food safety, colon ecosystem and colorectal carcinogenesis

F. Gueraud, C. Buisson, A. Promeyrat, N. Naud, E. Fouche, V. Bezirard, J. Dupuy, P. Plaisancie, C. Helies-Toussaint, L. Trouilh, J.-L. Martin, S. Jeuge, E. Keuleyan, N. Petit, L. Aubry, , F. H. PIERRE

This study evaluates the effects of sodium nitrite reduction, removal, and replacement in ham on microbiological safety, colon health, and colorectal cancer risk in a rat model.

bioRxiv · Mar 24, 2023 · PreprintPathology

Hepatocyte-specific deletion of Pparα promotes NASH in the context of obesity

M. Regnier, A. Polizzi, S. Smati, C. Lukowicz, A. Fougerat, Y. Lippi, E. Fouche, F. Lasserre, C. Naylies, C. Betoulieres, V. Barquissau, E. Mouisel, J. Bertrand-Michel, A. Batut, T. Al Saati, , A. Montagner

This study shows that deleting Ppar in hepatocytes promotes NASH in obese mice without glucose intolerance, highlighting the role of extra-hepatocyte PPAR in metabolic responses to HFD.

bioRxiv · Jan 18, 2019 · PreprintPathology