Author

A. Cotton

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Genetic & Genomic Medicine· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Approach:statistical model· 2 Approach:benchmark study· 1 Research Focus:benchmarking· 1 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

A. Adamo· 1 shared A. Enshaei· 1 shared A. Hamblin· 1 shared A. J. Mead· 1 shared A. Obermayer· 1 shared A. Rodriguez-Meira· 1 shared A. Tefferi· 1 shared A.-A. Olijnik-French· 1 shared

Activity by Year

2023-2023 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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Curved chords = mutual co-authorship

1 shared paperCancer Biology

A. Adamo

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Chromothripsis orchestrates leukemic transformation in blast phase MPN through targetable amplification of DYRK1A

C. K. Brierley, B. H. Yip, G. Orlando, H. Goyal, S. Wen, J. Wen, M. F. Levine, M. G. Jakobsdottir, A. Rodriguez-Meira, A. Adamo, M. Bashton, A. Hamblin, S. A. Clark, J. O'Sullivan, L. Murphi, , A. J. Mead

The paper identifies chromothripsis-induced amplification of DYRK1A in blast phase MPN as a driver of aggressive disease and a potential therapeutic target.

bioRxiv · Dec 8, 2023 · PreprintCancer Biology

Barriers to cascade screening in people at risk of Thoracic Aortic Disease: a mixed methods evaluation from the DECIDE-TAD initiative

Riccardo Giuseppe Abbasciano, Joanna Colleen Dionne, Joanne K. Miksza, Simon J. W. Oczkowski, Julian Barwell, Nora L. Shannon, R. Grant, Paul Clift, Riccardo Proietti, E. Hope, U. Ahern, K. Hewytt, L. Ghosh, R. Kaur, M. Lewis, , Gavin James Murphy

This study identifies barriers to cascade screening for thoracic aortic disease using mixed methods, finding low uptake due to awareness, health literacy, and cost concerns.

medRxiv · Dec 2, 2023 · PreprintGenetic & Genomic Medicine

CBFA2T3-GLIS2-dependent pediatric acute megakaryoblastic leukemia is driven by GLIS2 and sensitive to Navitoclax

M. Neault, C.-E. Lebert-Ghali, M. Fournier, C. Capdevielle, E. A. R. Garfinkle, A. Obermayer, A. Cotton, K. Boulay, C. Sawchyn, K. H. Nguyen, B. Assaf, F. E. Mercier, J.-S. Delisle, E. A. Drobetsky, L. Hulea, , F. A. Mallette

This study identifies GLIS2 as a driver of pediatric AMKL and shows its sensitivity to navitoclax, offering a potential therapeutic strategy.

Cell Reports · Sep 1, 2023Peer-reviewedCancer Biology