Author

A. Dumesnil

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Assay:bulk RNA-seq· 2 Research Focus:cancer & tumor microenvironment· 2 Research Focus:development & morphogenesis· 2 Organism:human· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1

Frequent Co-Authors

E. Brakenhielm· 3 shared C. Heron· 2 shared J.-B. Michel· 2 shared J.-P. Henry· 2 shared P. Mulder· 2 shared S. Fraineau· 2 shared S. Renet· 2 shared V. Richard· 2 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 42 mutual co-author links

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3 shared papersPhysiology

E. Brakenhielm

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Author profile →

3 papers

Recent·Most cited

Ezh2 as an epigenetic checkpoint regulator during monocyte differentiation: a potential target to improve cardiac repair after myocardial infarction

J. Rondeaux, D. Groussard, S. Renet, V. Tardif, A. Dumesnil, A. Chu, J.-P. Henry, Z. Badji, V. Claire, D. Beziau, D. Guerrot, M. Brand, V. Richard, E. Durand, E. Brakenhielm, S. Fraineau

This study shows that inhibiting EZH2 improves cardiac repair after myocardial infarction by enhancing monocyte repair functions and reducing cardiac dysfunction.

Nature Communications · Jul 25, 2023Peer-reviewedPhysiology

Blocking Interleukin-1β transiently limits left ventricular dilation and reduces cardiac lymphangiogenesis during pressure-overload in mice

C. Heron, T. Lemarcis, O. Laguerre, M. Bundalo, C. Valentin, M. Valet, A. Dumesnil, J.-B. Michel, P. Mulder, A. Zernecke, V. Tardif San Martin, E. Brakenhielm

This study shows that blocking IL-1β early after pressure-overload reduces left ventricular dilation but not heart failure, while late blocking reduces lymphangiogenesis without worsening fibrosis.

bioRxiv · Apr 1, 2023 · PreprintPhysiology

Regulation and impact of cardiac lymphangiogenesis in pressure-overload-induced heart failure

C. Heron, A. Dumesnil, M. Houssari, S. Renet, A. Lebon, D. Godefroy, D. Schapman, O. Henri, G. Riou, L. Nicol, J.-P. Henry, M. Pieronne Deperrois, A. Ouvrard-Pascaud, R. Hagerling, H. Chiavelli, , E. Brakenhielm

This study investigates cardiac lymphangiogenesis in pressure-overload-induced heart failure, finding that it limits inflammation and fibrosis, delaying heart failure development.

bioRxiv · Apr 27, 2021Physiology