Author

A. E. Marriott

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:drug response / drug discovery· 2 Research Focus:cell type annotation· 1 Research Focus:clustering· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

J. D. Turner· 3 shared A. Steven· 2 shared M. J. Taylor· 2 shared A. D. Davidson· 1 shared A. J. Russell· 1 shared A. Kipar· 1 shared A. Mansour· 1 shared A. Moorhead· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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3 shared papersMicrobiology

J. D. Turner

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

A dirofilariasis mouse model for heartworm preclinical research

A. E. Marriott, J. L. Dagley, S. Hegde, A. Steven, C. Fricks, U. DiCosty, A. Mansour, E. J. Campbell, C. M. Wilson, F. Gusovsky, S. A. Ward, W. D. Hong, P. O'Neill, A. Moorhead, S. McCall, , J. D. Turner

This paper establishes a mouse model for heartworm drug screening, showing it can effectively assess drug efficacy against D. immitis larvae.

bioRxiv · Apr 3, 2023 · PreprintMicrobiology

Structural requirements for dihydrobenzoxazepinone anthelmintics: actions against medically important and model parasites - Trichuris muris, Brugia malayi, Heligmosomoides polygyrus and Schistosoma mansoni

F. A. Partridge, C. J. Bataille, R. Forman, A. E. Marriott, J. Forde-Thomas, C. Häberli, R. L. Dinsdale, J. D. O'Sullivan, N. J. Willis, G. M. Wynne, H. Whiteland, J. Archer, A. Steven, J. D. Turner, J. Keiser, , D. B. Sattelle

The paper investigates the structure-activity relationship of dihydrobenzoxazepinone (DHB) compounds against various parasitic nematodes and trematodes, identifying key structural features for anthelmintic activity.

ACS Infectious Diseases · Apr 2, 2021Peer-reviewedPharmacology & Toxicology

Sequential infection with influenza A virus followed by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) leads to more severe disease and encephalitis in a mouse model of COVID-19.

J. J. Clark, R. Penrice-Randal, P. Sharma, A. Kipar, X. Dong, S. Pennington, A. E. Marriott, S. Colombo, A. D. Davidson, M. Kavanagh Williamson, D. A. Matthews, L. Turtle, T. Prince, G. Hughes, E. I. Patterson, , J. P. Stewart

This study shows that sequential infection with influenza A virus followed by SARS-CoV-2 in mice leads to more severe disease and encephalitis compared to single infections.

bioRxiv · Oct 13, 2020 · PreprintMicrobiology