Author

A. Foti

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Assay:bulk RNA-seq· 1 Organism:E. coli· 1 Organism:human· 1 Organism:rat· 1

Frequent Co-Authors

A. Biswas· 1 shared A. Prasse· 1 shared A. Zychlinsky· 1 shared B. Seeliger· 1 shared C. Johnson· 1 shared D. Humme· 1 shared E. Horbach· 1 shared G. H. Morales· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperImmunology

A. Biswas

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2 papers

Recent·Most cited

Tyrosine halogenation converts α-defensins into potent immunomodulators

A. Foti, R. Hurwitz, K. Textoris-Taube, M. Muelleder, A. Biswas, P. Fahjen, M. Winkler, U. Abu Abed, D. Humme, S. Thee, A. Prasse, B. Seeliger, M. Ralser, A. Zychlinsky

The paper identifies that tyrosine halogenation of α-defensins by hypohalous acids converts them into immunomodulatory mediators, revealing a novel oxidative mechanism in neutrophil function.

bioRxiv · Nov 1, 2023 · PreprintImmunology

Redox-mediated inactivation of the transcriptional repressor C3600 makes uropathogenic Escherichia coli exquisitely resistant to reactive chlorine species

S. Sultana, K. LeDoux, M. E. Crompton, O. Jankiewicz, G. H. Morales, C. Johnson, E. Horbach, K. P. Hoffmann, P. KR, R. Shah, G. M. Anderson, N. T. Mortimer, J. E. Schmitz, M. Hadjifrangiskou, A. Foti, J.-U. Dahl

This study identifies a redox-mediated mechanism in UPEC that enhances resistance to HOCl by inactivating the transcriptional repressor C3600 through disulfide bond formation.

bioRxiv · Aug 31, 2021 · PreprintMicrobiology