Author

A. Gerencser

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Organism:C. elegans· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. A. Shahmirzadi· 1 shared A. Foulger· 1 shared B. Schilling· 1 shared D. Wirtz· 1 shared F. Neri· 1 shared G. Lithgow· 1 shared J. Andersen· 1 shared J. Campisi· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperCell Biology

A. A. Shahmirzadi

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2 papers

Recent·Most cited

Senescent cell heterogeneity and responses to senolytic treatment are related to cell cycle status during cell growth arrest.

F. Neri, S. Zheng, M. Watson, P. Y. Desprez, A. Gerencser, J. Campisi, D. Wirtz, P.-H. Wu, B. Schilling

This study identifies functionally distinct senescent cell subpopulations based on cell cycle status, showing G2-arrested cells secrete more IL-6 and are more sensitive to senolytic treatment.

bioRxiv · Jun 22, 2024 · PreprintCell Biology

The Mitochondrial Permeability Transition Pore Activates the Mitochondrial Unfolded Protein Response and Promotes Aging

S. Angeli, A. Foulger, M. Chamoli, T. H. Peiris, A. Gerencser, A. A. Shahmirzadi, J. Andersen, G. Lithgow

This study shows that loss of OSCP in C. elegans activates the mPTP, triggers UPRmt, and shortens lifespan, while mPTP inhibition restores lifespan.

bioRxiv · Nov 3, 2020Cell Biology