Author

A. L. Cheung

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

N. P. Donegan· 2 shared A. F. Cowman· 1 shared A. R. Richardson· 1 shared B. Y. Zou· 1 shared D. A. Fidock· 1 shared D. H. Limoli· 1 shared D. Hutson· 1 shared D. Ma· 1 shared

Activity by Year

2019-2019 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersMicrobiology

N. P. Donegan

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Pan-active imidazolopiperazine antimalarials target the Plasmodium falciparum intracellular secretory pathway

G. M. LaMonte, D. S. Marapana, N. Gnadig, S. Ottilie, M. R. Luth, T. S. Worgall, F. Rocamora, G. M. Goldgof, R. Mohunlal, T. R. S. Kumar, J. K. Thompson, E. Vigil, J. Yang, D. Hutson, T. Johnson, , E. A. Winzeler

This study identifies imidazolopiperazines as targeting the Plasmodium falciparum secretory pathway by inhibiting protein trafficking and ER expansion.

bioRxiv · Aug 15, 2019Microbiology

The Toxin Antitoxin MazEF Drives Staphylococcus aureus Chronic Infection

D. Ma, J. B. Mandell, N. P. Donegan, A. L. Cheung, W. Ma, S. Rothenberger, R. M. Shanks, A. R. Richardson, K. L. Urish

This study shows that the MazEF toxin-antitoxin system in S. aureus inhibits biofilm formation and promotes antibiotic tolerance, facilitating the transition from acute to chronic infection.

bioRxiv · Jun 30, 2019 · PreprintMicrobiology

Interspecies signaling generates exploratory motility in Pseudomonas aeruginosa

D. H. Limoli, N. P. Donegan, E. A. Warren, A. L. Cheung, G. O'Toole

This study shows Pseudomonas aeruginosa changes its motility behavior in response to signals from Staphylococcus aureus, leading to directed movement and colony invasion.

bioRxiv · Apr 15, 2019Microbiology