Author

A. Thomson

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Physiology· 1 Plant Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 2 Organism:Arabidopsis· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:clinical cohort / trial· 1 Research Focus:host-pathogen & immune response· 1 Assay:imaging· 1

Frequent Co-Authors

G. A. Gray· 2 shared A. Caporali· 1 shared A. G. Rossi· 1 shared A. Johnson Dingee· 1 shared A.-M. Spiroski· 1 shared C. A. Tanney· 1 shared D. Ciren· 1 shared D. E. Newby· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersPhysiology

G. A. Gray

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Female Alms1-deficient mice develop echocardiographic features of adult but not infantile Alström Syndrome cardiomyopathy

E. J. McKay, I. Luijten, A. Thomson, X. Weng, K. Gehmlich, G. A. Gray, R. K. Semple

bioRxiv · Oct 16, 2023Physiology

Phosphorylation-dependent sub-functionalization of the calcium-dependent protein kinase CPK28

M. Bredow, K. W. Bender, A. Johnson Dingee, D. R. Holmes, A. Thomson, D. Ciren, C. A. Tanney, K. E. Dunning, M. Trujillo, S. C. Huber, J. Monaghan

This study identifies a phosphorylation site in CPK28 that differentially regulates its function in immune homeostasis and stem elongation in Arabidopsis.

bioRxiv · Oct 16, 2020Plant Biology

Eosinophil deficiency promotes aberrant repair and adverse remodelling following acute myocardial infarction

I. S. Toor, D. Ruckerl, I. Mair, R. Ainsworth, M. Meloni, A.-M. Spiroski, J. M. Felton, A. Thomson, A. Caporali, T. Keeble, K. Tang, A. G. Rossi, D. E. Newby, J. E. Allen, G. A. Gray

This study shows that eosinophil deficiency after myocardial infarction leads to adverse cardiac remodeling, which can be mitigated by IL-4 therapy.

bioRxiv · Sep 9, 2019Immunology