Author

D. Ruckerl

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 5

Signature Assays, Organisms & Methods

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Organism:mouse· 5 Approach:experimental assay· 4 Research Focus:cell type annotation· 3 Assay:bulk RNA-seq· 2 Research Focus:cell signaling & trafficking· 2 Research Focus:differential expression· 2 Research Focus:host-pathogen & immune response· 2 Approach:statistical model· 2

Frequent Co-Authors

J. E. Allen· 3 shared R. Sahputra· 2 shared A. Caporali· 1 shared A. G. Rossi· 1 shared A. Thomson· 1 shared A. Woolf· 1 shared A.-M. Spiroski· 1 shared D. E. Newby· 1 shared

Activity by Year

2019-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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3 shared papersImmunology

J. E. Allen

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5 papers

Recent·Most cited

Monocyte-derived peritoneal macrophages protect C57BL/6 mice against surgery-induced adhesions

R. Sahputra, K. Dejyong, A. Woolf, M. Mack, J. E. Allen, D. Ruckerl, S. Herrick

This study shows that monocyte-derived peritoneal macrophages reduce surgery-induced adhesions in C57BL/6 mice, while BALB/c mice show increased adhesions with reduced monocyte influx.

bioRxiv · Jul 12, 2022 · PreprintImmunology

Post-transcriptional regulatory feedback encodes JAK-STAT signal memory of interferon stimulation

E. Kalliara, M. Kardynska, J. Bagnall, D. G. Spiller, W. Müller, D. Ruckerl, S. K. Biswas, J. Smieja, P. Paszek

The paper shows that interferon stimulation induces long-term STAT1 desensitization through post-transcriptional feedback, enabling cells to remember past stimulation and adjust future responses.

bioRxiv · May 13, 2022Immunology

The magnitude of airway remodelling is not altered by distinct allergic inflammatory responses in BALB/c vs C57BL/6 mice but matrix composition differs

J. E. Parkinson, S. Pearson, D. Ruckerl, J. E. Allen, T. E. Sutherland

bioRxiv · Oct 20, 2020Immunology

Eosinophil deficiency promotes aberrant repair and adverse remodelling following acute myocardial infarction

I. S. Toor, D. Ruckerl, I. Mair, R. Ainsworth, M. Meloni, A.-M. Spiroski, J. M. Felton, A. Thomson, A. Caporali, T. Keeble, K. Tang, A. G. Rossi, D. E. Newby, J. E. Allen, G. A. Gray

This study shows that eosinophil deficiency after myocardial infarction leads to adverse cardiac remodeling, which can be mitigated by IL-4 therapy.

bioRxiv · Sep 9, 2019Immunology

The essential role played by B cells in supporting protective immunity against Trichuris muris infection is dependent on host genetic background and is independent of antibody

R. Sahputra, D. Ruckerl, K. Couper, W. Muller, K. Else

This study shows B cells support protective immunity against T. muris in C57BL/6 mice but not BALB/c mice, independent of antibodies and dependent on genetic background.

bioRxiv · Feb 18, 2019 · PreprintImmunology